Typically, Estrogen Dominance is due to a combination of one or more of the following: Chronic stress. Poor gut/liver health. Environmental toxins known as xenoestrogens.
Body fat: Fat tissue (adipose tissue) secretes estrogen. Having a high percentage of body fat can lead to high estrogen levels. Stress: Your body produces the hormone cortisol in response to stress. Producing high amounts of cortisol in response to stress can deplete your body's ability to produce progesterone.
Eat a diet high in cruciferous vegetables, including cabbage, broccoli, cauliflower, and Brussels sprouts. Consider supplementing indole-3-carbinol 300 mg daily if inadequate dietary intake. 5. Eat a diet high in whole soy foods which help to shift estrogen metabolism toward increased levels of 2-hydroyestrone.
Estrogen dominance can affect a woman's body in many ways, including abnormal menstruation (heavy/painful periods), PMS, headaches, decreased sex drive, bloating, mood swings, fatigue, anxiety & depression, breast tenderness, endometriosis, fibroids, and hormonal weight gain.
Health conditions: Some health conditions have an association with or lead to estrogen dominance. These include polycystic ovary syndrome (PCOS), uterine fibroids, endometriosis, and certain cancers. Insulin resistance also increases estrogen levels.
It's very important to know which estrogen dominance foods to avoid. You should steer clear of red meats, processed food, refined carbs, and foods rich in saturated fats. These categories of foods have a negative impact on the body in general.
When estrogen is too high or too low you may get menstrual cycle changes, dry skin, hot flashes, trouble sleeping, night sweats, vaginal thinning and dryness, low sex drive, mood swings, weight gain, PMS, breast lumps, fatigue, depression and anxiety.
Magnesium promotes healthy estrogen clearance
By supporting the COMT enzyme (catechol-o-methyltransferase) in the liver, magnesium promotes the healthy excretion of estrogen (9). This may reduce the risk of the estrogen excess conditions (such as fibroids) associated with low COMT function (10).
In 2 months: You can improve estrogen metabolism and reduce PMS symptoms. Estrogen dominance is at the root of most hormonal conditions, so flushing the excess hormones from your body is essential to maintaining your overall health.
The same goes for stress as the body uses up more progesterone to manufacture the stress hormone cortisol and in turn leaves us with an excess of estrogen.
What foods cause high estrogen? Foods that reportedly increase estrogen include flax seeds, soybean products, chocolate, fruit, nuts, chickpeas, and legumes. Before we delve into why these foods are said to increase estrogen, we need to look at two important definitions; phytoestrogens and lignans.
Fatigue
Unfortunately, estrogen dominance can cause fatigue. This symptom can occur whether or not you have insomnia. If you experience persistent tiredness or feel more drained of energy than usual, talk to your doctor about being tested for hormonal issues.
A Fred Hutchinson Cancer Research Center study involving postmenopausal, overweight, and obese women who took 2,000 IUs of vitamin D daily for a year found that those whose vitamin D blood levels increased the most had the greatest reductions in blood estrogens, which are a known risk factor for breast cancer.
Consuming 200 milligrams or more of caffeine per day-roughly equivalent to two cups of coffee-can either increase or decrease a woman's estrogen level, depending on her ethnic background and the source of the caffeine, a study from the National Institutes of Health (NIH) finds.
Research shows that avocados can help reduce the absorption of estrogen and boost testosterone levels. They also improve heart health and aid in satiety. We need enough healthy fats to make hormones, and avocados are a great source of hormone building blocks.
The foods you need to avoid include: flax seeds, dried fruits, sesame seeds, garlic, peaches, berries, wheat bran, tofu, tempeh, dairy products, meat, alcohol, grains, and legumes.