High protein intake elevates IGF-1 levels and promotes growth, which could accelerate the onset of puberty. Poor diet. Children with lower-nutrient diets tend to enter puberty earlier. A diet rich in processed foods and meats, dairy, and fast food is disruptive to normal physical development.
Children who ate a lot of animal protein at that age (meat, eggs, and dairy) started puberty a year earlier than those who ate a lot of plant-based protein. Children eating a lot of vegetable protein started puberty 7 months later on average, and children eating a lot of animal protein started puberty 7 months earlier.
Causes of early puberty
a problem in the brain, such as a brain tumour. damage to the brain as a result of an infection, surgery or radiotherapy. a problem with the ovaries or thyroid gland. a genetic disorder, such as McCune-Albright syndrome – read more about McCune-Albright syndrome on the MedlinePlus website.
Fact: There is no scientific evidence that drinking milk causes early puberty. In the United States, girls are entering puberty at younger ages than they were in the first half of the 20th century. Researchers do not know the exact trigger for early-onset puberty, although it is likely influenced by many factors.
According to the National Institutes of Health, puberty usually begins in girls between 8 and 13 years of age, and in boys between 9 and 14 years of age. Puberty is considered to be early in boys before age 8 and girls before 9 years old. This is sometimes called “precocious puberty.”
This treatment, called GnRH analogue therapy, usually includes a monthly injection of a medication, such as leuprolide acetate (Lupron Depot), or triptorelin (Trelstar, Triptodur Kit), which delays further development. Some newer formulations can be given at longer intervals.
Healthy foods have nutrients that are important for growth and development during puberty. Teenagers should limit salty, fatty and sugary foods, low-fibre foods, and drinks with caffeine or a lot of sugar.
Conversely, a higher vegetable protein intake at 3–4 and 4–5 years might delay pubertal development. Additionally, we demonstrated that children with a higher dietary quality in the years preceding ATO, defined using a nutrient density-based diet index, entered puberty at a later age.
Poor diet. Children with lower-nutrient diets tend to enter puberty earlier. A diet rich in processed foods and meats, dairy, and fast food is disruptive to normal physical development. Exposure to EDCs (endocrine-disrupting chemicals).
For most children, early puberty starts for no known reason. It can run in families. Sometimes there is a problem in the brain, such as an injury, a tumor or an infection. Early puberty can also be caused by a problem in the sex glands (testes or ovaries), the thyroid gland or the adrenal glands.
to be linked to a suppression of the activity of neurons involved in releasing a hormone that triggers the ovulation process (menstruation). The researchers concluded that these results suggest that vitamin D may inhibit early pubertal onset and/or the rapid progression of puberty.
Consumption of junk food tends to increase which affects nutritional improvement. This is because the fat content, animal protein and trans fat contained in junk food will trigger the release of hormones that affect the occurrence of menarche and the emergence of secondary signs in children faster than normal age.
Delayed puberty can be caused by an underlying medical condition (e.g., celiac disease or a hormone deficiency). There's often a hereditary component to delayed puberty. If a parent was late in starting puberty, it's more likely that his or her child might be, too.
Rarely, certain conditions, such as infections, hormone disorders, tumors, brain abnormalities or injuries, may cause precocious puberty. Treatment for precocious puberty typically includes medication to delay further development.
Providers can treat precocious puberty—marked by breast development before age 8 or testes growth before age 9—with hormonal suppressants, also called puberty blockers. With supervision, these reversible drugs safely and effectively delay a child's development until they're ready.
Researchers and physicians hypothesized about possible causes for the increase in early puberty, such as increasing rates of obesity; greater exposure to endocrine-disrupting chemicals found in food, plastics, and personal-care products; and stressful or abusive home environments.
Avoid exposure to hormones.
Keep your children away from medications, creams, and lotions that contain reproductive hormones like estrogen and testosterone. This is the best way to avoid early puberty, as it cannot be prevented in most cases.
The most dangerous age is 14. If you know any teenagers this might not come as a surprise, but research has confirmed that risk-taking peaks during this exact moment in mid-adolescence.
Growth hormones found in meat could have a substantial effect on prepubescent children. If a child isn't yet producing growth hormones themselves, consumption of these growth hormones through either meat or dairy products could enter the child into puberty around seven months earlier, a study has found.
During puberty, boys need about 2,800 calories a day, while girls need about 2,200 calories per day, including plenty of protein and nutrients like zinc, to develop and grow. Consider magnesium, a mineral that plays a role in over 300 essential metabolic reactions.
Sugar. Excess sugar consumption may also affect the height of kids. Sugar increases the insulin level in the body and prevents the body from growing properly.
Untreated precocious puberty usually leads to short stature and can also cause significant emotional and behavioral issues.
Low vitamin D levels have been found in girls with precocious puberty, as well, although the exact relationship between vitamin D deficiency and early development remains unclear.
IGF-1 modulates the onset of puberty and pubertal progression by stimulating the GnRH25). So, it is conceivable that vitamin D-mediated effects may influence IGF-1 levels and pubertal onset through an effect on gonadotropin and sex hormone.