Severe Serratia infection (bacteremia) carries a mortality rate of 26%.
marcescens have been associated with serious outcomes. The overall mortality rate of S. marcescens bacteremia remains high, ranging from 25-58%.
Prognosis of Serratia infection is generally good in urinary tract infections, pneumonia, and local wound infections.
SURVIVAL OUTSIDE HOST: S. marcescens may survive from 3 days to 2 month on dry, inanimate surfaces, and 5 weeks on dry floor 16. The organism may survive less than 4 days in a blood bag under aerobic conditions and 20 days in semi-anaerobic/anaerobic conditions 17.
Serratia marcescens is a rare agent causing peritonitis, and few cases of Serratia marcescens peritonitis in previous reports were successfully cured without removing the peritoneal catheter.
Once established, the organism usually cannot be eliminated entirely. However, periodic and thorough cleaning of the surfaces where the pink slime occurs, followed by disinfection with chlorine bleach, appears to be the best way to control it.
marcescens can be a harmful bacterium. Serratia marcescens is now known to be a common cause of human infections in the respiratory tract, digestive tract, and in wound site infection. Serratia marcescens is a common cause of so-called hospital acquired infections in both patients and healthcare workers.
Optimal growing conditions for Serratia marcescens include dampness, mineral deposits and a climate ranging from warm to room temperature. It feeds on the minerals, fatty deposits and residue from your soap and personal hygiene products.
Serratia is usually treated with antibiotics, but it is naturally resistant to several common antibiotics such as ampicillin, macrolides, and first-generation cephalosporins. Antibiotics used to treat Serratia include: Cefepime (Maxipime) Broad-spectrum beta-lactam/Beta-lactamase inhibitor combinations.
Serratia marcescens is an opportunistic pathogen that is primarily responsible for hospital-related infections. You can get infected with Serratia marcescens through: Contact with contaminated hospital equipment, such as catheters. Direct contact through infected people.
It is commonly found in the respiratory and urinary tracts of hospitalized adults and in the gastrointestinal systems of children.
Serratia marcescens (S. marcescens) is a gram-negative bacillus that occurs naturally in soil and water and produces a red pigment at room temperature. It is associated with urinary and respiratory infections, endocarditis, osteomyelitis, septicemia, wound infections, eye infections, and meningitis.
Serratia infections should be treated with an aminoglycoside plus an antipseudomonal beta-lactam, as the single use of a beta-lactam can select for resistant strains. Most strains are susceptible to amikacin, but reports indicate increasing resistance to gentamicin and tobramycin.
Symptoms may include fever, frequent urination, dysuria, pyuria, or pain upon urination. In 90% of cases, patients have a history of recent surgery or instrumentation of the urinary tract.
The main risk factors for bacteraemia/sepsis which is caused by Serratia is hospitalization, placement of intravenous catheters, intraperitoneal catheters and urinary catheters and prior instrumentation of the respiratory tract [2–5]. Serratia marcescens causes both opportunistic and nosocomial infections.
The good news is Serratia marcescens is mostly harmless. Touching it while you're showering or cleaning won't cause any problems (in rare cases it can cause infections through open wounds or the eye).
marcescens were only killed by the use of chloramphenicol at ten and one hundred times concentrations used to kill planktonic bacteria, non-other of the antibiotics tested had the same effect. These suggest that chloramphenicol might be utilized for ALT against not only S.
this various incubation period, the results elucidated that 48 hours incubation showed the highest pigment production in S.
Conclusions: Treatment of SM infections should include carbapenems or aminoglycosides in combination with third-generation (and eventually fourth-generation) cephalosporin. Cotrimoxazole should be considered in cases of uncomplicated urinary infections.
Filtering water can remove particles that may contain Serratia marcescens. Water filtration systems can help filter the chlorine so that the “pink stuff” won't be on sinks. Banish pink slime from your home today!
It produces a fishy-urinary odor (trimethylalamine). It can be isolated from food, soil, water, plants, insects, and sewage. It is a potential pathogen for insects, animals, and humans and is an important cause of nosocomial (hospital-acquired) infections in humans.
What Causes Pink Mold in a Shower? Serratia marcescens and Aureobasidium pullulans are the most common bacteria that cause pink mold in a bathroom. These airborne bacterial species love moist environments like showers, where they feed on minerals and fatty deposits in soap and shampoo residue.
S. marcescens are inherently resistant to a range of narrow-spectrum penicillins including ampicillin, amoxicillin, amoxicillin-clavulanate, ampicillin-sulbactam and several narrow-spectrum cephalosporins (101) (Table 1). This resistance is attributed to the presence of a chromosomal AmpC beta-lactamase enzyme.
Serratia marcescens, which can cause nosocomial outbreaks,and urinary tract and wound infections, is abundant in damp environments (Figure). It can be easily found in bathrooms, including shower corners and basins, where it appears as a pink–orange–red discoloration, due to the pigment known as prodigiosin.